Semaglutide

In research settings, Semaglutide is studied for its potential to: 1-Enhance insulin secretion in a glucose-dependent manner, aiding glycemic control in metabolic models. 2-Suppress glucagon release, helping regulate postprandial glucose spikes. 3-Delay gastric emptying, which may support prolonged satiety and reduced caloric intake. 4-Reduce food intake and body weight, through central appetite-suppressing mechanisms via the hypothalamus. 5-Support beta-cell function and preservation, making it a promising candidate in type 2 diabetes models.

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Description

Disclaimer: This is a research chemical only. It’s not a medication, and it’s not approved for human or animal use outside of authorized studies

Sequence H‑His‑Aib‑Glu‑Gly‑Thr‑Phe‑Thr‑Ser‑Asp‑Val‑Ser‑Ser‑Tyr‑Leu‑Glu‑Gly‑Gln‑Ala‑Ala‑Lys(ε‑AEEAc‑AEEAc‑γ‑Glu‑17‑carboxyheptadecanoyl)‑Glu‑Phe‑Ile‑Ala‑Trp‑Leu‑Val‑Arg‑Gly‑Arg‑Gly‑OH (Semaglutide)
Molecular Formula C₁₈₇H₂₉₁N₄₅O₅₉
Molecular Weight ~4113.6 Da
PubChem CID 56843331
CAS # 910463‑68‑2

Semaglutide is a long-acting GLP-1 receptor agonist under extensive investigation for its role in regulating appetite, blood glucose levels, and energy balance. Structurally modified for enhanced half-life and resistance to enzymatic degradation, Semaglutide mimics endogenous glucagon-like peptide-1 (GLP-1), a hormone central to glucose metabolism and satiety signaling.

Its extended duration of action allows for once-weekly experimental administration, making it favorable in long-term metabolic and endocrine research protocols.

Additional information

MG

5 MG, 10 MG, 20 MG

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